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Progress of enteral nutrition support and intestinal microecological adjustment in surgical patients with gastric cancer
Chen Jie, Lian Bo, Li Mengbin, Zhao Qingchuan
Department of Digestive Surgery, Xijing Hospital of Digestive Diseases, Air Force Medical University, Xi′an 710032, Shaanxi, China
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Abstract  Gastric cancer is the fifth malignant tumor in incidence and the third in mortality worldwide. For gastric cancer patients, the main treatment plan is still surgical resection. Due to psychological factors, tumor consumption, mechanical obstruction, increased catabolic metabolism and other factors of gastric cancer will lead to serious nutritional metabolism problems among patients, it will affect the safety of surgery and postoperative recovery. Surgery is the main treatment for gastric cancer, but the trauma, physiological changes and postoperative chemotherapy may further aggravate the above problems. Intestinal microecology is an important part of human immune function. However, existing studies suggest that tumor metabolism, surgery and chemotherapy may cause changes in the intestinal flora. With the development of the concept of accelerated rehabilitation surgery, the comprehensive treatment of gastric cancer has attracted more and more attention from surgeons. How to improve the nutritional status of patients with gastric cancer after surgery, reduce intestinal flora disorder, and thus reduce postoperative complications and accelerate postoperative recovery has become the focus of gastrointestinal surgeons. This paper reviews the application of perioperative enteral nutrition support and intestinal microecological regulation in patients with gastric cancer.
Key wordsGastric cancer      Nutritional treatment      Intestinal microecological regulation      Enhanced recovery after surgery     
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Chen Jie
Lian Bo
Li Mengbin
Zhao Qingchuan
Cite this article:   
Chen Jie,Lian Bo,Li Mengbin, et al. Progress of enteral nutrition support and intestinal microecological adjustment in surgical patients with gastric cancer[J]. Electron J Metab Nutr Cancer, 2020, 7(1): 22-26.
URL:  
http://182.92.200.144/EN/10.16689/j.cnki.cn11-9349/r.2020.01.005     OR     http://182.92.200.144/EN/Y2020/V7/I1/22
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